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Extra resources for Annual Review of Immunology Volume 29 2011

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1999. Mice lacking expression of secondary lymphoid organ chemokine have defects in lymphocyte homing and dendritic cell localization. J. Exp. Med. 189:451–60 76. Nakano H, Gunn MD. 2001. Gene duplications at the chemokine locus on mouse chromosome 4: multiple strain-specific haplotypes and the deletion of secondary lymphoid-organ chemokine and EBI-1 ligand chemokine genes in the plt mutation. J. Immunol. 166:361–69 77. Schumann K, Lammermann T, Bruckner M, Legler DF, Polleux J, et al. 2010. Immobilized chemokine fields and soluble chemokine gradients cooperatively shape migration patterns of dendritic cells.

Org by University of Iowa on 05/26/11. For personal use only. b AIRE or Deaf-1 TSA Subset eTACS UEA-1+ stromal cells FRC LEC MHC class II + + – – AIRE + – – – Deaf-1 ? + + + Figure 5 Schematic representation of antigen-presenting stromal cells. (a) Antigenpresenting stromal cells likely express TSA via the activity of AIRE or Deaf-1, resulting in presentation of tissue-restricted self-antigens in MHC class I. If stromal cell subsets positive for MHC class II were capable of autophagy, they could present self-antigens in MHC class II.

J. Exp. Med. 193:621– 30 25. Mebius RE, Rennert P, Weissman IL. 1997. Developing lymph nodes collect CD4+ CD3− LTβ+ cells that can differentiate to APC, NK cells, and follicular cells but not T or B cells. Immunity 7:493– 504 26. Meier D, Bornmann C, Chappaz S, Schmutz S, Otten LA, et al. 2007. Ectopic lymphoid-organ development occurs through interleukin 7-mediated enhanced survival of lymphoid-tissue-inducer cells. Immunity 26:643–54 27. Schmutz S, Bosco N, Chappaz S, Boyman O, Acha-Orbea H, et al.

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