By P. J. Schwartz (auth.), Prof. Dr. Ronald W. F. Campbell, Prof. Dr. Michiel J. Janse (eds.)
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Extra info for Cardiac Arrhythmias: The Management of Atrial Fibrillation
J w > ci z 0 u •• 12 cm 50 Sem AFIB. 50 REFR. PER. (ms) 150 Fig. 2. Relation between induction of atrial fibrillation and refractory period, conduction velocity and wavelength of the initiating premature beat. Because wavelength is the product of refractory period and conduction velocity "iso-wavelength" curves are drawn at 8 cm and 12 cm. Because of the effects of a wide variety of administered drugs, a wide range of refractory periods and conduction velocities was achieved. ) were obtained over a wide range of conduction velocities and refractory periods.
Force of contraction (Fe) was recorded at the apex of the preload active tension curve via the inductive force displacement transducer in conjunction with a HeUige frequency carrier preamplifier. The transmembrane potential was detected intracellularly by the use of 10 to 20-MQ glass microelectrodes filled with KCI 3 mol/I. The signals were led off by means of a voltage follower with input capacitance compensation. For the determination of maximal upstroke velocity of the action potential, dVidt was obtained by analogue differentiation.
We compard APDs measured at different right atrial sites in patients with normal sinus rhythm and patients who were cardioverted from atrial fibrillation. Patients pro ne to atrial fibrillation exhibited marked differences in APD between different atrial sites. On average, patients with recurrent atrial fibrillation had significantly greater heterogeneity (or dispersion) of atrial APDs during sinus rhythm than the control group. Thus, it would make sense to treat atrial fibrillation with drugs that prolong the action potential.